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In nerves exposed to a hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose further vitality substrate is required when the tissue vitality demand will increase. Such data are essential as they show that glycogen has an necessary position below physiological conditions. The truth that glycogen supported CAP conduction in the absence of exogenously utilized glucose probably has no value in ascribing a role to glycogen, since aglycaemia is an artificial experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus cannot be glycogen’s essential role. However supplying lactate to axons when tissue energy demand increases implies a novel position for mind glycogen and suggests it acts as an vitality buffer fairly than an power reserve, whose position is not to single-handedly help conduction, but to act in live performance with ambient ranges of normoglycemic glucose.
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